ALZHEIMER'S & DEMENTIA
Alzheimer’s & Dementia
Good News for people suffering from Alzheimer’s or Dementia
Vascular dementia as a diagnosis was essentially forgotten back in 1968 when pathologist Leopold Litz did a study of demented patients at a VA hospital. He found that most of these vascular dementia patients were, in fact, suffering from Alzheimer’s disease. However, his work was based solely on the presence of Alzheimer’s-linked neurofibrillary tangles, etc. and he did not examine the blood vessels and their endothelium closely, mainly because the available technology and the physical problems inherent in doing this type of study precluded this. From that time on almost all dementia cases have been thrown into the category or rubric “Alzheimer’s”, though this is dead wrong IMO. Most Alzheimer’s patients suffer from endothelial damage of their cerebral blood vessels and often have nocturnal hypoxemia as well.
Virtually no neurologist evaluates suspected Alzheimer’s patients using a nocturnal oximetry test which in my opinion is very important for this simple reason: episodes of nocturnal hypoxemia invariably lead to the development of amyloid plaque in vital areas of the brain. Just putting these patients on night time oxygen can at times bring them back to normal – as long as they use it consistently. Chelation therapy will reverse Alzheimer’s pathology in about 60% of the cases. However, the only way this effect can be maintained is for chelation to be done every 3 weeks. The reason this works so well probably lies in the fact it decreases the patient’s red cell calcium content. Old blood vessels and their endothelial damage injures the red cell membranes and this allows calcium to accumulate within them. This increase in calcium causes rigidity of RBCs such that they cannot readily move through narrowed and diseased blood vessels which culminates in compromised tissue oxygenation.
Chelation removes the calcium from RBCs which means they can better transport oxygen for about 3 weeks after each chelation treatment. Stem cells help by fixing the endothelial damage and in this regard I have seen good results with these as well. The use of autologous adult stem cells is actually the easiest method of treating truly demented people since few of them can sit still for a 3 hour chelation iv. In addition: Heavy metals are often a part of the AD-generating equation as are chronic infections, all of which needs to be properly diagnosed and dealt with. Hormonal imbalances and addition of HGH, IGF-I, PRP, autologous conditioned serum, cerebrolysin, DMSO, methylcobalamin, folic acid, vitamins etc. all can helpful as well. In mild cases if you apply all of these there is a high probability of reversing the disease in almost 100% of AD sufferers.
This success rates drops to 80% or so in moderate cases and to 20% in advanced cases (The quantity of autologous adult stem cells used by most practitioners to treat severe AD is likely far lower than what is needed to effect remediation and a clinical turnaround. In all likelihood, massive, repeated doses are needed though no one has done this as part of any clinical trial that I am aware of.
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Vascular Contributions to Cognitive Impairment and Dementia
A Statement for Healthcare Professionals From the American Heart Association/American Stroke Association
Vascular contributions to cognitive impairment and dementia are important. Understanding of VCI has evolved substantially in recent years, based on preclinical, neuropathologic, neuroimaging, physiological, and epidemiological studies. Transdisciplinary, translational, and transactional approaches are recommended to further our understanding of this entity and to better characterize its neuropsychological profile. There is a need for prospective, quantitative, clinical-pathological-neuroimaging studies to improve knowledge of the pathological basis of neuroimaging change and the complex interplay between vascular and Alzheimer disease pathologies in the evolution of clinical VCI and Alzheimer disease. Long-term vascular risk marker interventional studies beginning as early as midlife may be required to prevent or postpone the onset of VCI and Alzheimer disease. Studies of intensive reduction of vascular risk factors in high-risk groups are another important avenue of research.
I bring this to my site to point out again that vascular (i.e. blood vessel disease) is a major component of Alzheimer’s and/or dementia and should be treated as soon as possible. I have developed a new unique formula for the treatment of vascular disease so if anyone wants to be a researcher and help determine if my product is better than anything else out there let me know and I will give it away for my direct out of pocket costs for the first 100 or so people who need treatment of their hypertension, kidney failure, atherosclerosis, dementia, and or Alzheimer’s.
Thanks. - Dr. S