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EDTA CHELATION

An essential part of regaining your health, staying healthy and getting ready for stem cell therapy is chelation! To understand why this is true a little background is needed:

There are two general types of poisons that affect our bodies – heavy metals and chemicals. Heavy metals include lead, mercury, cadmium, etc. which react with oxygen, glucose, etc. to produce free radicals which damage tissues where the metals are embedded in the body.
Metals such as copper, zinc, iron, etc. are usually a part of the structures of proteins in living organisms. If these metals are released into tissues, they react with oxygen and form free radicals. Heavy metals such as lead and mercury sabotage the body’s free radical mop up system which basically keeps them from forming molecules with unpaired electrons. These free radical electrons then can react with other molecules and form a chain reaction of destruction. The result is that your tissue “spoils” or becomes “rancid”. These “rancid” membranes are a major part of aging and disease. One of the biggest challenges in reversing aging and chronic disease processes is to actually reverse this “rancidity” of diseased tissues. Chelation removes the basic cause behind the formation of “rancid” membranes by removing the free metals that bring about the formation of “rancid” oxidized cell membranes.
EDTA chelation is endorsed for the treatment of atherosclerotic vascular disease by the American College for Advancement in Medicine (ACAM), comprising of 750 licensed physicians. Its proponents claim that EDTA consistently improves blood flow and relieves symptoms associated with atherosclerosis in over 80% of the patients treated. In early studies, it was observed that patients being treated for acute lead intoxication with EDTA and who had cardiovascular disease showed improved stress tolerance and less chest pain with physical exertion. In 1956 Norman Clarke published an article in the American Journal of Medical Science on the successful treatment of patients with severe angina pectoris through the use of EDTA.

In 1988, Drs. Olszewer and Carter reported on the treatment of 2870 patients with EDTA chelation therapy. Over 93% of the patients who suffered from narrowed coronary arteries showed good to excellent improvement. 97% of those with narrowed leg arteries showed improvement and 60% of patients with narrowed brain arteries showed increased circulation from EDTA chelation.

More than one million patients have received over twenty million infusions to-date. No significant adverse effects have been reported when the ACAM protocol has been followed. Early reports of renal injury and more recent adverse effects resulted from excessive doses being given (greater than 50 mg/Kg/day), from infusions that were too fast (less than three hours), and from failing to replace calcium, magnesium and other nutrients that are recommended in the ACAM protocol.

The benefits of EDTA chelation are proposed to include:

  • Binds with calcium ions in arterial walls and plaque. With a decrease in the extracellular quantities of calcium, the arterial wall becomes more compliant, less rigid and more elastic. This results in a greater delivery of blood to each of the body’s organs in instances in which the arterial compliance had been previously compromised.
  • Binds with extracellular calcium (outside the cell) which induces calcium withdrawal from inside injured artery wall cells. With the removal of calcium, the cell is better able to resume normal energy production, waste removal and functioning.
  • Binds with heavy metals such as lead, mercury, cadmium, aluminum, uranium, etc. These metals block enzyme activity and their removal helps restore enzyme functions within the arterial walls.
  • Binds to iron and copper ions, transition metals that promote free radical reactions. By removing these metal ions, EDTA chelation slows or stops free radical damage. This includes reductions in lipid peroxidation and oxidized cholesterol.
  • EDTA has an “anti-sticky” effect on blood platelets. Lipid peroxides and free radicals inhibit the synthesis of prostacyclin. Reducing these influences helps to normalize the balance between prostacyclin levels and thromboxane, and reduce the risk of “sticky” platelets and blood clots that can block an artery.
  • EDTA has a membrane fluidizing effect on erythrocytes, making them more flexible and better able to maneuver through small capillaries. This results in improved tissue oxygenation.
  • EDTA uncouples disulfide and mineral cross-links, enabling greater flexibility in connective tissue.
  • EDTA may inhibit NF kappa B which plays a pivotal role in programmed cell death.
  • EDTA’s chelation of zinc and copper may reverse the cortical deposition of amyloid beta involved in Alzheimer’s disease.

In summary, arteries become hardened and non-elastic as atherosclerosis develops. EDTA chelation therapy “softens” these hardened arteries by reducing the collagen and elastin cross linkages by removing the metals that are generating the cross-linking free radicals.

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